Publication & Citation Trends
Publications
0 total
Human-Relevant Mechanisms and Risk Factors for TAK-875-Induced Liver Injury Identified via a Gene Pathway-Based Approach in Collaborative Cross Mice. OA
Cited by 12
Semantic Scholar
New Phenotypic Cytotoxicity Assay for ROS-inducing Compounds using Rat Renal Epithelial Cells.
Cited by 1
Semantic Scholar
Polygenic architecture informs potential vulnerability to drug-induced liver injury OA
Cited by 71
Semantic Scholar
Quantitative Systems Toxicology Analysis of In Vitro Mechanistic Assays Reveals Importance of Bile Acid Accumulation and Mitochondrial Dysfunction in TAK-875-Induced Liver Injury OA
Cited by 40
Semantic Scholar
Fasiglifam (TAK-875) Alters Bile Acid Homeostasis in Rats and Dogs: A Potential Cause of Drug Induced Liver Injury OA
Cited by 48
Semantic Scholar
Navigating tissue chips from development to dissemination: A pharmaceutical industry perspective OA
Cited by 77
Semantic Scholar
Chemically-Induced Hepatocarcinogenesis
Cited by 1
Semantic Scholar
Understanding drug-cytokine synergistic toxicity OA
Cited by 0
Semantic Scholar
Research Topics
Carcinogens and Genotoxicity Assessment
(28)
Pharmacogenetics and Drug Metabolism
(17)
Drug-Induced Hepatotoxicity and Protection
(16)
Glutathione Transferases and Polymorphisms
(16)
Drug Transport and Resistance Mechanisms
(15)
Affiliations
University of Wisconsin System
National Center for Toxicological Research
DuPont (United States)
United States Food and Drug Administration
University of Wisconsin–Madison