Publication & Citation Trends
Publications
0 total
V-domain Ig Suppressor of T cell Activation Expression During Hemorrhage or Sepsis-Induced Acute Respiratory Distress Syndrome: Insights From a Mouse Model. OA
Cited by 2
Semantic Scholar
Differential Effects of Endothelial Cell- as opposed to Neutrophil-Lineage Restricted PD-L1 Gene Expression on Experimental Murine Shock/ Sepsis-Induced Lung Injury
Cited by 0
Semantic Scholar
VISTA functions as a protective immune checkpoint in indirect acute respiratory distress syndrome by modulating systemic and compartmentalized inflammation
Cited by 0
Semantic Scholar
invariant Natural Killer T Cells Modulate the Peritoneal Macrophage Response to Polymicrobial Sepsis.
Cited by 2
Semantic Scholar
Herpes virus entry mediator signaling blockade produces mortality in neonatal sepsis through induced cardiac dysfunction OA
Cited by 1
Semantic Scholar
Severity of critical illness correlates with CD3-low expression on iNKT-cells among septic surgical patients
Cited by 0
Semantic Scholar
Lymphocyte HVEM/BTLA co-expression after critical illness demonstrates severity indiscriminate upregulation, impacting critical illness-induced immunosuppression OA
Cited by 3
Semantic Scholar
SPLENIC INVARIANT NATURAL KILLER T CELLS PLAY A SIGNIFICANT ROLE IN THE RESPONSE TO POLYMICROBIAL SEPSIS
Cited by 3
Semantic Scholar
Research Topics
Immune Response and Inflammation
(112)
Sepsis Diagnosis and Treatment
(72)
Cell death mechanisms and regulation
(32)
Immune Cell Function and Interaction
(27)
Immune cells in cancer
(24)
Affiliations
National Institutes of Health
John Brown University
University of Rhode Island
Providence College
Brown University