Publication & Citation Trends
Publications
0 total
Loss of Smooth Muscle &agr;-Actin Leads to NF-&kgr;B–Dependent Increased Sensitivity to Angiotensin II in Smooth Muscle Cells and Aortic Enlargement OA
Cited by 54
Semantic Scholar
Abstract 458: Loss of Smooth Muscle α-actin in Mice Results in Thoracic Aortic Aneurysms via Increased Reactive Oxygen Species, Increased Nox4,and Increased Angiotensin II type 1 Receptor-Mediated Signaling
Cited by 0
Semantic Scholar
Loss of fi bulin-4 disrupts collagen synthesis and maturation : implications for pathology resulting from EFEMP 2 mutations
Cited by 6
Semantic Scholar
Loss of fibulin-4 disrupts collagen synthesis and maturation: implications for pathology resulting from EFEMP2 mutations. OA
Cited by 53
Semantic Scholar
Abnormal mechanosensing and cofilin activation promote the progression of ascending aortic aneurysms in mice OA
Cited by 50
Semantic Scholar
MMP17/MT4-MMP and thoracic aortic aneurysms: OPNing new potential for effective treatment. OA
Cited by 10
Semantic Scholar
Fibulin-4 and fibulin-5 in elastogenesis and beyond: Insights from mouse and human studies. OA
Cited by 143
Semantic Scholar
Angiotensin-Converting Enzyme–Induced Activation of Local Angiotensin Signaling Is Required for Ascending Aortic Aneurysms in Fibulin-4–Deficient Mice OA
Cited by 59
Semantic Scholar
Research Topics
Connective tissue disorders research
(17)
Aortic aneurysm repair treatments
(13)
Aortic Disease and Treatment Approaches
(10)
Protease and Inhibitor Mechanisms
(5)
Cell Adhesion Molecules Research
(4)
Affiliations
Karlsruhe Institute of Technology
University of Tsukuba
Southwestern Medical Center
Maine Farmland Trust
Institute of Molecular Medicine