Publication & Citation Trends
Publications
0 total
Targeting Wnt/β-Catenin and circadian regulator restores PRC2/EZH2 controlled chromatin bivalency and suppresses cell state diversity.
Cited by 0
Semantic Scholar
Abstract 1359: Cooperativity between DNMT and EZH2 activity drives neuroendocrine phenotype in advanced prostate cancer.
Cited by 0
Semantic Scholar
Abstract PR027: Spatial heterogeneity atlas of prostate cancer evolution (SHAPE): Spatial and genomic landscapes of advanced prostate cancer with and without Lu-PSMA therapy
Cited by 0
Semantic Scholar
Abstract PR035: Cooperativity between DNA methylation and EZH2 activity drives neuroendocrine phenotype in advanced prostate cancer
Cited by 0
Semantic Scholar
Abstract PR022: Polycomb dysregulation shapes chromatin bivalency critical for prostate cancer lineage plasticity
Cited by 0
Semantic Scholar
Crosstalk between EZH2 and DNA methylation mediates neuroendocrine prostate cancer lineage plasticity
Cited by 0
Semantic Scholar
Epigenetic De-repression of PROX1 Promotes Neuroendocrine Prostate Cancer Progression.
Cited by 1
Semantic Scholar
PD35-05 COOPERATIVITY BETWEEN DNA METHYLATION AND EZH2 ACTIVITY DRIVES NEUROENDOCRINE PHENOTYPE IN ADVANCED PROSTATE CANCER
Cited by 0
Semantic Scholar
Intraindividual epigenetic heterogeneity underlying phenotypic subtypes of advanced prostate cancer
Cited by 5
Semantic Scholar
Targeting the tumour cell surface in advanced prostate cancer
Cited by 21
Semantic Scholar
Research Topics
Prostate Cancer Treatment and Research
(62)
Glutathione Transferases and Polymorphisms
(27)
Estrogen and related hormone effects
(22)
Mechanisms of cancer metastasis
(21)
Microtubule and mitosis dynamics
(20)
Affiliations
Cleveland State University
Broad Institute
Boston University
Cleveland Clinic
Harvard University