Publication & Citation Trends
Publications
0 total
Caspase-6-Resistant Mutant Huntingtin Does not Rescue the Toxic Effects of Caspase-Cleavable Mutant Huntingtin in vivo OA
Cited by 8
Semantic Scholar
Rescue from excitotoxicity and axonal degeneration accompanied by age-dependent behavioral and neuroanatomical alterations in caspase-6-deficient mice. OA
Cited by 80
Semantic Scholar
Low Levels of Human HIP14 Are Sufficient to Rescue Neuropathological, Behavioural, and Enzymatic Defects Due to Loss of Murine HIP14 in Hip14−/− Mice OA
Cited by 13
Semantic Scholar
Natural history of disease in the YAC128 mouse reveals a discrete signature of pathology in Huntington disease.
Cited by 78
Semantic Scholar
Altered palmitoylation and neuropathological deficits in mice lacking HIP14. OA
Cited by 122
Semantic Scholar
Mice lacking caspase-2 are protected from behavioral changes, but not pathology, in the YAC128 model of Huntington disease OA
Cited by 35
Semantic Scholar
Cleavage at the 586 Amino Acid Caspase-6 Site in Mutant huntingtin Influences Caspase-6 Activation In Vivo OA
Cited by 122
Semantic Scholar
Phosphorylation of Huntingtin at Ser421 in YAC128 Neurons Is Associated with Protection of YAC128 Neurons from NMDA-Mediated Excitotoxicity and Is Modulated by PP1 and PP2A OA
Cited by 93
Semantic Scholar
Research Topics
Genetic Neurodegenerative Diseases
(15)
Mitochondrial Function and Pathology
(14)
Cholesterol and Lipid Metabolism
(14)
Drug Transport and Resistance Mechanisms
(10)
Peroxisome Proliferator-Activated Receptors
(9)
Affiliations
Leiden University
Children's Hospital of Philadelphia
University of British Columbia
Medical University of South Carolina
Universität Hamburg