Publication & Citation Trends
Most Cited Works
Publications
11 total
Mice carrying a R142C<i>Notch 3</i>knock-in mutation do not develop a CADASIL-like phenotype
Cited by 42
OpenAlex
Mice carrying a R142C Notch 3 knock–in mutation do not develop a CADASIL‐like phenotype
Cited by 44
Semantic Scholar
Characterization of <i>Notch3</i>‐deficient mice: Normal embryonic development and absence of genetic interactions with a <i>Notch1</i> mutation
Cited by 258
OpenAlex
Characterization of Notch3‐deficient mice: Normal embryonic development and absence of genetic interactions with a Notch1 mutation
Cited by 278
Semantic Scholar
A CADASIL-mutated Notch 3 receptor exhibits impaired intracellular trafficking and maturation but normal ligand-induced signaling
Cited by 117
OpenAlex
Notch signaling is involved in the regulation of Id3 gene transcription during Xenopus embryogenesis
Cited by 43
OpenAlex
The origin of the ankyrin repeat region in Notch intracellular domains is critical for regulation of HES promoter activity
Cited by 93
OpenAlex
Volume contents OA
Cited by 0
Semantic Scholar
Research Topics
Developmental Biology and Gene Regulation
(7)
Cerebrovascular and genetic disorders
(4)
Neurogenesis and neuroplasticity mechanisms
(2)
Cell Adhesion Molecules Research
(2)
Congenital heart defects research
(2)
Frequent Co-Authors
Affiliations
University of Queensland
Karolinska Institutet